Cow Nutritional Metabolism - Prevention of Hypomagnesemia

2, hypomagnesemia

2.1 Causes

There are two factors that cause bovine hypomagnesemia, one is the role of exogenous factors, that is, the content of magnesium in feed diet is insufficient, which is the main cause of the disease; the other is endogenous factor The role, that is, chemical changes in the ruminal content of cattle caused malabsorption of magnesium. Due to the lack or reduction of magnesium in cattle, the imbalance of magnesium, potassium, calcium, and phosphorus causes the main cause of the disease. Grass that grows after raining is rich in protein and potassium, but contains lower levels of magnesium, calcium, sodium, and sugar, so it is a specific time of onset in late spring, summer, and early autumn, especially when it is very rich in organic nitrogen (such as The grazing on the land where Ammonium bicarbonate is applied and the fattening of the grass in the orchard field are more susceptible to disease. In addition, reduced consumption of cattle can affect the absorption of magnesium; cattle have diarrhea can increase the discharge of magnesium. Calving cows during lactation due to calf sucking milk, reduce the cow's body magnesium and calcium content, generally about 7 days postpartum cows onset.

2.2 symptoms

2.2.1 The acute type of cattle that are grazing suddenly stretches to the back of one side and assumes a lateral anti-bow position, rolling left and right, repetitively appearing ankylosing spasm for 1 minute or so, during which time the jaws are closed and the nystagmus is closed. Palpate foam, raise your ears, and retract your eyes. After a few moments of quietness, she relapsed and strenuously struggled. His body temperature reached 40 to 40.5°C, breathing and pulse accelerated, and his heart beats. He could be heard a few steps away. It usually takes less than 1 to 3 hours to die without treatment.

2.2.2 Subacute mental depression and gait. The sensitivity to light and sound contact is enhanced within 24 h. Ears erected, nystagmus, and blister. Frequent urination, defecation, weakened rumen movements, decreased milk production. The muscles of the head (especially the nose, upper lip), abdomen, and extremities tremored, and then a tetanus-like ankylosing paralysis occurred. Blood tests are characterized by a sharp drop in serum magnesium values ​​to 0.4 to 0.9 mg/100 mL (normal values ​​1.8 to 3.0 mg/dL). The affected animal may recover within a few days, or it may become chronic type, lying on the ground, with stiff limbs, good response to magnesium preparations, but it may recur.

2.2.3 The chronic type does not show clinical symptoms except for a drop in blood magnesium concentration, and some may also appear unresponsive, unwilling to exercise, and have no choice for feeding.

2.3 Prevention

In the dangerous period of the disease, oral magnesium oxide or magnesium sulfate, 30 ~ 40 g per cow. A certain amount of hay is given before the animal is grazing in early spring; it is not advisable to overgraze or eat too much when the grass is flourishing. In areas with magnesium deficiency, green pastures may be sprayed with 20 to 30 kg of magnesium sulfate per hectare of pasture before cattle or sheep are grazing or harvesting silage to prevent the occurrence of this disease.

Treatment with calcium and magnesium mixture (250 g calcium gluconate, magnesium sulfate 50 g, dubbed 1 000 mL injection) 400 ~ 800 mL intravenous injection; with 25% magnesium sulfate solution 200 mL intramuscular injection, can also be 2 ~ 4 g Magnesium sulphate dissolved in 5% glucose solution 1 000 mL slowly intravenously. After the symptoms improved, they were replaced with the maintenance dose of intramuscular injection; 10% calcium gluconate 500 mL intravenously, and then 20% magnesium sulfate or magnesium chloride 200 ~ 400 mL subcutaneous injection. At the same time, 60 g of magnesium chloride was taken orally for at least 1 week, and then gradually stopped; 15 g of magnesium sulfate, 40 g of calcium gluconate, 50 g of glucose, and 500 mL of purified water were intravenously injected after sterilization.

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