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Cucumber Sclerotinia has a wide range of host hosts. Apart from harming cucumbers, it can also harm tomatoes, sweet peppers, eggplants, peas, potatoes, carrots, celery, and various cruciferous vegetables, which mainly occur in the middle tube sheds and adjacent buildings. The greenhouse is cultivated on a cucumber.
Hazardous symptoms Cucumber sclerotia disease mainly damages stem base and fruit, but also can harm stems and leaves. It can occur from the seedling stage to the adult stage of cucumber. Stem disease, the main part of the disease at the base of the stem and the stem of the tiller, the initial occurrence of water-soaked spots, light brown after expansion, diseased stems soft rot longitudinal crack, diseased stems and leaves above vines withered. When the humidity is high, the disease minister produces a layer of white cotton flocculent mycelia. After injury, the medullary part of the stem is damaged. At the end of the disease, it is rotten and hollow, and the white mycelia and black sclerotia are peeled off. The sclerotium is mucus-like, round or irregular, early white, and later becomes black outside and white inside. The fruit was infected and the disease started to rot in the umbilicus of young fruit. The fruit surface grew white cotton flocculent mycelium and formed black granular sclerotia. The leaves were infected and they were initially immersed in water spots. After expansion, they were grayish-brown and nearly round and large. The edges were not obvious. The diseased part was soft rot and white cotton wool-like mycelium was produced.
Prevention measures 1. Seed treatment. Before sowing, soaking in warm water at 50°C for 10 minutes, immediately move into cold water, cool, dry and germinate after sowing, then the sclerotia mixed in the seeds can be killed.
Second, clean up the pastoral. In time, the old leaves were removed and the remaining flowers on the fruit were removed. The diseased plants were found to be promptly removed or cut to remove diseased fruits, and brought out of the shed to be burned or buried deeply. After the harvest, the diseased body was completely removed, and the soil was deeply turned to prevent sclerotia from sprouting.
Third, the mouth rotation. With aquatic vegetables, grasses and onions and garlic vegetables every year round.
Fourth, strengthen management. Closely plant the plant and control the temperature and humidity in the shelter shed and the multi-storied greenhouse to protect the greenhouse, release air in a timely manner, and especially prevent the humidity in the night shed from rising rapidly. This is a key measure to prevent and cure this disease. Pay attention to reasonable control of the amount of water and fertilizer, watering time in the morning, and open the shed in time to reduce the humidity within the shed. In particular, in the spring before the invasion of the cold, it is necessary to timely cover the small arch plastic film, and cover the grass around the booth to prevent plants from freezing.
Fifth, the seedbed is controlled regularly and timely. Before the transplanting, the seedlings must be transplanted with the drug, not to transplant the seedlings and weak seedlings, and strictly control the transplanting of the seedlings.
Sixth, soil disinfection. Per square meter with 50% carbendazim powder 8 ~ 10 grams, and dry fine soil 10 ~ 15 kg after mixing and spread disinfection source.
Seven, chemical control. In the initial stage of onset of spraying, spray every 7 to 10 days, and even spray 3 to 4 times, depending on the severity of the disease as the development of the field, if necessary, increase the frequency of spraying. Pharmacy can use 43% Haldex suspension 2000-3000 times (40-50 grams per acre), 50% quickan wettable powder 1000 times (100 grams per acre), 50% acetaminophen Wettable powder 1000 times liquid (100 grams per acre), 50% carbendazim wettable powder 800 times (125 grams per acre), 70% thiophanate-methyl 1000 times (100 doses per acre g) and so on.
Measures for Controlling Cucumber Sclerotinia
Experts pointed out that the sclerotinia of cucumber is caused by the asphyxial subphylum A. sclerotiorum. The bacterium nucleus in the soil and in the residual tissues of the diseased plants and mixed in the seeds for winter or summer. Sclerotia generally survive for about two years. When the environmental conditions are suitable, the sclerotia germinates to produce the ascitium plate, and the ascospore sporadically released from the ascitic plate spreads and spreads through the air flow. The senescent leaves or unpeeled petals are infiltrated and directly penetrated through the stratum corneum to cause the initial impregnation. The invading pathogen destroys the cells and tissues of the host, spreads and destroys tissues that have not been infected with pathogens, and repeats the infection through contact between diseased and healthy plants. The diseased leaf is in contact with the healthy leaves or stems, and the diseased petals fall on the healthy leaves. The pathogenic bacteria can expand and cause the diseased stems and leaves to develop.